It means she has a blood coagulation(clotting) problem.
People with deep vein thrombosis have this problem and the usual treatment is warfarin. I wouldnt advise using any natural treatments for this problem as regular monitoring by a Dr is required.
Now being the risk taker I am before I went a step further I would try a course of MMS. I have seen photos of how the blood cells taken with dark field microscopy greatly improve after using mms. What does this suggest? To me it suggests that the blood cells, both red and white, (and why not platelets to?) have a microbe invasion and these microbes could be in her bone marrow also and this is why she has these blood dyscrasias.
You say huh? if there were microbes why do they not know it and why do they not give her antibiotics?
Until conventional medicine allows for the use of dark field microscopes and until they start treating for these unseen microbes the deception will continue and many many more will die that don't need to.
You ask the doctor what causes these problems and he will say he doesn't know or he will say that this part or that part is not working correctly or he will say its autoimmune but still he will not be able to tell you what causes these parts to not work right.
__________________ "The nurse should be cheerful, orderly, punctual, patient, full of faith, - receptive to Truth and Love" Mary Baker Eddy
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The Anticardiolipin AB (Antibody Syndrome) (Lupus Anticoagulant) is caused by an antibody response against phospholipid (a major component of the cell wall). The antibody response results in a heterogenous group of clinical conditions including blood clots, stroke, heart attack, low platelet count, spontaneous abortions, and vague neurologic symptoms.
This abnormality was first identified as a prolongation of the clotting test (the PTT) in persons who had systemic lupus erythematosus, therefore the name lupus anticoagulant was coined. Although there is a high incidence of this in persons with lupus, it occurs in many persons without lupus and does not predict future development of lupus. Although some persons have prolongation of clotting tests, they do not bleed but are instead prone to the development of blood clots.
Persons with this disorder may be asymptomatic or have various syndromes. Affected persons may have recurrent blood clots involving the heart and resulting in a heart attack, involving the vessels to the brain and resulting in a stroke or other neurologic symptoms, or involving the vessels feeding the developing fetus in pregnant women resulting in miscarriage. The blood clots may also occur in the extremities (deep vein thrombosis) or lung (pulmonary embolus).
How Do You Get It??
There is no evidence to suggest that this disorder is inherited and the cause of it is not known.
Who Knows If They Have It??
Testing for this disorder may be done by clotting tests specifically designed to detect the lupus anticoagulant or by evaluating for the presence of the antibody. These blood tests are done by a blood sample that requires special handling in a coagulation laboratory for accurate diagnosis. to diagnose this disorder, two tests must be done on different occasions because the antibody may wax and wane. The clotting test is inaccurate in persons on blood thinners or with underlying bleeding disorders.
The lupus anticoagulant/ anticardiolipin antibody test may be positive in persons with infections, using antibiotics, cancers, or other medications and usually returns to normal when the infection is cleared or the medication stopped.
What Should You Do About It??
The optimum treatment for these patients is not known. Persons with the abnormality who have had a deep vein thrombosis are at increased risk of developing recurrent blood clots and should be maintained on blood thinners. Persons with other manifestations may benefit from aspirin like agents with or without the addition of blood thinners.
Women with the abnormality who become pregnant may be at increased risk of miscarriage and should be managed with medication and close monitoring during pregnancy.
Factor 5 and 8 are genes that further affect coagulation.
anticardiolipin, AB, factor 5 and factor 8
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