Obesity is defined as weight that exceeds 15 percent of normal weight for height and body type. "Morbid" obesity exceeds 20 percent of optimum weight. The long-term health implications are well known, in fact, obesity is considered an outright disease. Life expectancy may be decreased in overweight and obese individuals. An obese or overweight person is at high risk for a number of serious health problems, including heart disease, high blood pressure, stroke, varicose veins, dementia, psychological stress, depression, osteoarthritis, high cholesterol, allergies, psoriatic arthritis, and diabetes.
Omega-3 is an essential fatty acid that is deficient in the diets of many Americans. In the late 1970s, scientists learned that the native Inuits in Greenland, who consumed a diet very high in omega-3 fatty acids, had surprisingly low rates of heart attacks. Since that time, more than 4,500 studies have been conducted in an attempt to understand the beneficial roles that the omega-3 fatty acids play in human metabolism and health.
Scientists from the Fred Hutchinson Cancer Research Center and the University of Alaska-Fairbanks performed a study to examine whether high eicosapentaenoic (EPA) and docosahexaenoic (DHA) acid intakes could modify associations of obesity with chronic disease risk. This cross-sectional study involved 330 Yup'ik Eskimos with an average age of 45 years and that 70 percent of them were overweight or obese. Red blood cell fatty acids were measured and found increases in C-reactive protein (CRP) and triglyceride levels in obese Eskimos with low omega-3 blood levels while such increased levels were not observed in people with high blood levels of EPA and DHA. The researchers commented �Our findings may have important clinical relevance for the prevention of some obesity-related diseases. Obesity prevalence in the US and worldwide has been increasing over the past decades, with subsequent increases in rates of diabetes and other obesity-associated diseases. It is likely that these associations are partly mediated by the positive associations of obesity with triglycerides and CRP, two biomarkers that strongly and independently predict risks of CVD and possibly diabetes.�1
1 Makhoul Z, Kristal AR, Gulati R, et al. Makhoul Associations of obesity with triglycerides and C-reactive protein are attenuated in adults with high red blood cell eicosapentaenoic and docosahexaenoic acids. Eur J Clin Nutr. Mar2011.
There is another route by which omega 3's EPA and DHA may reduce the rate of obesity and that is by reducing inflammation in the brain.
Omega 3's are anti inflammatory.
Adipose tissue is pro inflammatory.
The more inflammatory your digestive system and body is the more likely you will have pathogenic obese type gut flora. Making omega 3 intake a priority and reducing the intake of pro inflammatory omega 6 industrial seed oils will tip the balance towards the anti inflammatory side and the lean type gut flora that do not promote fat storage nor extract as many calories from the food you consume (nor make you fart as much)
Omega 3's by reducing the body's inflammatory status reduce depression and one aspect of depression in many people is comfort eating. Resolving the inflammation associated with depression will reduce the tendency to snacking and comfort eating.
One feature of dieting is the requirement to use will power to control portion size/frequency of eating/and regulate activity levels. The demands of most dieting regimes oblige the participant to exert will power/self control and this inevitably raises cortisol levels. Cortisol, is by definition a corticosteroid and the prolonged upregulation or oral administration of corticosteroid's inevitably produces side effects. You can read them yourself but it should be fairly obvious that the more you demand self control/will power and shame people into weight loss by making them feel bad about themselves then the more counterproductive this approach will be because the side effects of raised cortisol levels are according to the UK NHS
Side effects of oral corticosteroids that are used on a short-term basis include:
an increase in appetite,
fluid retention, and
mood changes, such as feeling irritable, or anxious.
The more you make people feel crap about being overweight the less likely they are to diet successfully.
One aspect of overeating I've not talked about is addiction. Junk foods are processed in such a way as to enhance the additive potential of those so called foods. Your ability to maintain any any anti addiction strategy is determined by your omega 3 status. People with higher omega 3 status are far more likely not to fail when it comes to substance abuse Because giving up junk foods requires exactly the same self control as giving up other addictive substances it follows correcting omega 3 anti inflammatory status at the same time makes the whole process a lot easier and less likely to fail.
I don't doubt that fatty acids prevent heart attacks. They also protect eyes, joints, and probably every cell in the body in one way or another, however, preventing obesity? ...
Obesity is caused by overeating, especially simple carbs over a long period of time, and can be triggered by stress, depression, and probably a million other reasons, but until this is controlled, obesity will continue to be a major health problem no matter the intake of omega 3.
As long as fast food, soda, and other processed junk foods are available, obesity will continue to rise, and at some point will bankrupt the system, which is already spending over 2 trillion per year on sick care.
Taking omega 3 fatty acids is great, but is only one spoke in the wheel.
Let Food Be Your Medicine And Medicine Be Your Food.(Hippocrates)
This in the context of insufficient exercise. The question we have to ask is WHY do people feel the need to eat when they have a stored reserve of calories about their person and WHY are they so lethargic.
especially simple carbs over a long period of time,
We all can understand why low carbohydrate diets work the problem is why can't~won't "official health professional advisors understand common sense. There are NO ESSTENTIAL CARBOHYDRATES so WHY are they the base of the diet pyramid or a major component of the Eatwell plate
Where are the evidence based peer reviewed studies showing they help people stay healthy.
Doesn't the whole history of "healthy eating advice " since 1975 show an exsalation in obesity, cancer, diabetes and Alzheimer's.
Insanity is doing the same thing over and over again and expecting a different result." Einstein
and can be triggered by stress, depression, and probably a million other reasons, but until this is controlled, obesity will continue to be a major health problem no matter the intake of omega 3.
Most of the reasons that trigger obesity, such as stress, depression or what have you involve inflammation (if you disagree then provide non inflammatory examples) therefore correcting inflammatory status, of which omega 3 rebalancing is a key factor (alongside and in synergy with, magnesium, vitamin d3, and melatonin.) is fundamental to success and a requisite to prevent weight regain.
Remember The Role of Impaired Mitochondrial Lipid Oxidation in Obesity Mary Madeline Rogge
explains the role of dsyfunctional mitochondria in OBESITY. i cannot understand how any program for weight maintainance can be successful if the problems associated with dysfunctional mitochondria are ignored.
As long as fast food, soda, and other processed junk foods are available, obesity will continue to rise,
Indeed it's the case that when doctors recommend the ideal nutrition to ensure weight gain they recommend products like Ensure Plus Corn, sugar, omega 6 oils and Genetically modified soy. + a few chemicals to finish the job.
The ultimate Junk food in a drink.
and at some point will bankrupt the system, which is already spending over 2 trillion per year on sick care.
Yes Obesity related Diabetes, cancer Alzheimer's and other diet related conditions is already breaking the bank and it simply isn't sustainable.
Taking omega 3 fatty acids is great, but is only one spoke in the wheel.