You can read the report
here
Bear in mind the list of panel members.
A. Catharine RossProfessor of Nutrition and Occupant of Dorothy FoehrHuck Chair in Nutrition, Pennsylvania State University
Steven A. Abrams Professor of Pediatrics, Baylor College of Medicine
John F. Aloia Professor, SUNY at StonyBrook, Chief Academic Officer, Winthrop-University Hospital
Patsy M. BrannonProfessor, Division of Nutritional Sciences, Cornell University
Steven K. Clinton Professor, Division of Hematology and Oncology, The Ohio State University
Ramon A. Durazo-Arvizu Associate Professor, Loyola University Stritch School of Medicine
J. Christopher Gallagher Professor of Medicine, Creighton University Medical Center
Richard L. Gallo Professor of Medicine and Pediatrics, University of California–San Diego
Glenville Jones Head, Department of Biochemistry and Professor of Biochemistry & Medicine, Queens University, Ontario
Christopher S. Kovacs Professor of Medicine (Endocrinology), Memorial University of Newfoundland
Joann E. Manson Professor of Medicine and the Elizabeth Brigham Professor of Women’s Health, Harvard Medical School
Susan T. Mayne Professor of Epidemiology and Public Health, Yale School of Public Health
Clifford J. Rosen Senior Scientist, Maine Medical Center Research Institute
Sue A. Shapses Professor, Department of Nutritional Sciences, Rutgers University
How many leaders in the field of vitamin D research are included?
Well there are a few names I recognise but those are not the people I would regard as lead researchers or the most knowledgeable in Vitamin D research.
Ask a panel of secondraters to produce a report and who would expect anything but a second rate report.
Rather than place the onus on those proposing to restore historically higher vitamin d status than those currently achievable, given the increasing amounts of ozone in urban and industrialized agriculture, the onus of proof should be on establishing that the lower level currently obtainable under ozone polluted atmospheres are indeed safe.
Given that year on year 25(OH)D levels are declining the boot has to be on the other foot.
What evidence do the panel have for supposing that human breast milk is not meant to be vitamin D replete at 60ng/ml or that babies should not benefit from vitamin D replete breast milk?
Given that below 40ng/ml the human body does not store vitamin D3 why do the panel suppose not having a reserve store of D3 (as happens at 60ng/ml) to last through the winter does not give those people a natural advantage in immune function?
Only when we have studies comparing those maintaining 25(OH)D levels above 50ng/ml with those below 30ng/ml will we know for certain but in order to demonstrate the benefits those levels will have to be sustained over several years. It's also no good thinking it's a one size fits all scenario. We know perfectly well that at any daily vitamin D intake the response is around 60ng/l 150ng/ml from the lowest to the highest responders. So we have to 25(OH)D test at least twice yearly everyone on the trial to ensure the minimum status 30ng/ml or 50ng/ml are sustained through the year and over the whole extent of the trial.
I'm not putting my life at risk of unnecessary infections or cancer by opting to be in the 30ng/ml group. If I can stay in the 60ng/ml group I'll happily participate.
But I am not convinced by that group of losers.
PS I should perhaps say that I've not quarrel with their calcium findings. I've been concerned about calcium intakes for some time and I doubt anyone needs more than 600mg/daily calcium in addition to food/water sources. I can't see why most people cannot obtain sufficient calcium from food sources. It's a totally different position than vitamin D for which food sources can only provide 10% of daily need and we can't rely on laying naked in the midday sun if that was socially acceptable.