"Phlebotomy of humans with impaired glucose tolerance and ferritin values in the highest quartile increased adiponectin improved glucose tolerance. These findings demonstrate causal role for iron as risk factor for metabolic syndrome and role for adipocytes in modulating metabolism through adiponectin in response to iron stores".
Iron overload for women starts after menopause donating from them will slow the ageing process.
Men should start donating earlier,
In the UK regular donation from 68~70 allows you to continue donating over 70 yrs old so is particularly useful for preventing/delaying Alzheimer's.
Why on earth UK NHSBlood donation service don't explain the recent science relating to the BENEFITS of donating blood to those doing the donating is beyond belief and tantamount to medical negligence.
Bloodletting... isn't that how president Washington died?
On the other hand, iron is the one thing you don't want too much of. If you have your iron tested and the doctor says you are borderline low, that is exactly where you want to be, and I never suggest taking multivitamins that contain iron in any amount.
Donating blood is probably your best option for lowering iron, and extra vitamin C will help you absorb more iron from the leafy green veggies that you eat, so taking iron supplements is dangerous and should only be done in rare circumstances when your body is very low.
Women generally live longer than men, and the only logical reason is because they menstrate and we don't. (why is it called menstrate?)
...Then there is menopause..
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Let Food Be Your Medicine And Medicine Be Your Food.(Hippocrates)
I'm not sure how we went from blood draining to diabetes, however, I can tell you iron is not the cause. It may be a factor or trigger, but its not the cause. If it was, every man over 50 would be diabetic.
There are other triggers as well, including vaccines, viruses, grains, and several other immune supressors, however, there are only two things that have changed over the last century that could explain the "explosion" of diabetes. First, we consume an average of 150 pounds of sugar per person in North America, along with 55 pounds of corn syrup. When you consider most health minded people don't consume sugar or corn syrup, along with babies, and most diabetics, the average becomes much higher.
In other words, the actual amount of sugar and corn syrup consumed is probably double the above figures when you allow for the correct population adjustment.
Secondly, the biggest change that has taken place over the last 100 years is light... that is artificial light. We were meant to sleep from sundown to sunrise, however, with all kinds of artificial lighting, our internal clocks become confused, and hormones such as melatonin are not produced at the right times.
This causes chaos within the body. Melatonin protects us from all kinds of diseases, including cancers...and since we repair while asleep, the whole system crashes, especially for people who work shiftwork or otherwise remain awake late at night for long periods.
This will eventually cause big changes in metabolism, thus weight gain, insulin resistance, and disease.
The body cannot repair and protect itself without the raw materials that it needs.
Iron as the cause? not likely. If that were the case, diabetes could easily be avoided by taking antioxidants.
Don't we need cholesterol for our skin and our brains and organs? Isn't cholesterol good?
But the article was saying
Quote:
A study of 60 overweight people found that bloodletting reduced blood pressure, as well as levels of 'bad' LDL cholesterol and increased 'good' HDL cholesterol
It's the high number of small particles that cause the damage not actually the total amount of cholesterol in your blood.
this Peter Attia series explains in greater detail
but you can have a HIGH particle count and LOW CHOLESTEROL and be at risk and you can have a LOW particle count and HIGH CHOLESTEROL and be at the lowest risk.
So looking just at a TOTAL CHOLESTEROL count may give a misleading impression of risk.
On the other hand, as blood volume was not replaced following phlebotomies, patients on multi-drug therapy or with type 2 diabetes might also have a dysfunctional endothelium and sympathetic response to relative hypovolemia. They might be unable to compensate for hypovolemia as healthy donors do. Indeed, most previous evidence were from cohort and cross-sectional studies of healthy donors and* from high-ferritin T2DM patients and carriers of hereditary hemocromatosis in whom blood volume was restored to normal at each procedure