\r\n \r\n I have talked about the benefits of Blood donation in this thread in some detail
\nCan draining blood cut cholesterol and ward off cancer?
\nBut it\'s always good to remind people and hope someone reads the thread and actually acts on that information.
\n
\nYou in the USA are luckier because you can donate more regularly than we in the UK. For some reason our NHS BLOOD DONATION service refuse to acknowledge that there is any benefit to the donors in donation.
\n
\nIt\'s totally ridiculous the staff all have been trained to be obsequious to the point it makes you cringe.
\n
\nThey don\'t seem to grasp the simple idea that they are providing a free anti-ageing service.
\n
\nThey seem too stupid to realise that when you donate blood your blood pressure drops. If they encouraged people with high blood pressure to donate more often they may save a fortune on blood pressure lowering medications.
\n
\nDiabetes is raging in the UK with more people being diagnosed and treated year on year.
\nWe all know high transferrin levels associate with 2~3 times more diabetes incidence yet NO ONE ever points out the obvious fact that regularly donating blood keeps iron, ferritin and transferrin levels lower.
\n
\nThere should be a poster at every blood donation clinic explaining the benefits of regular blood donation to the donors. (together of course with information about ensuring your do stay iron replete)
\n
\n
\nImage from Join-Hands. org\r\n

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\r\n \r\n How often can you donate in the UK?
\n
\nAnother great benefit is reduction of chronic inflammation (which ironically, eventually can cause anemia!)
\n
\nUnfortunately, in Mexico (where I live now) and the US hypertension is an exclusion criterion for donation!
\nAs I mentioned, most blood banks never check ferritin and are so stupid that many if not most of the donors are menstruating women, whose ferritin is below 30 and who should never donate.
\n
\nAll great cultures practiced phlebotomy, the Chinese, Egyptians, Persians, Greeks, Romans, Aztect, Inca, the British Empire, etc, unfortunately, like all therapies, some misused or abused it and it became taboo.
\n
\nFerritin is an invaluable diagnostic tool that not only blood banks, but most doctors seldom or never request. I have found internal bleeding, early stage rheumatoid arthritis, lupus and many cases of hemochromatosis thanks to it. I request chemistry 24, cytometry, general urine test, ekg, ferritin and insulin from every patient (all of that costs about 62 U$ on the saturday special in a nation-wide lab in Mexico).
\n
\n
\n
\nA caveat about blood donation. Common deficiencies such as Cu, Mg, Si, etc, are excerbated, unless supplements are taken.\r\n

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\r\n \r\n In UK Male donors can donate 4 times in 12 months with a minimum interval of 12 weeks between donations. Female donors donate every 16 weeks to reduce the risk of iron deficiency.
\n
\nHowever in most towns the blood donation teams visit every 16 weeks so if male you have to make appointments online at different towns as the teams that come round aren\'t connected to the internet so can only make appointments for that centre so in practice most donors only donate at 16 week intervals.
\nFor NEW DONORS there is an age limit Only if you have donated between the ages of 68 and 70 are you permitted to continue donating over the age of 70 so it\'s important when approaching 70 to ensure you don\'t forget to keep donating or you could lose the chance.\r\n

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\r\n \r\n Uric acid levels are related directly to iron and molybdenum levels and inversely to copper and magnesium levels and kidney function. Moroever, uric acid levels over 5 increase the risk of hypertension, gout, diabetes, osteoarthritis, cardiovascular disease, kidney damage (which in turn raises urate levels), etc,
\n
\nAlthough medical texts state that hypertension is idiopathic (of unknown cause) they do recommend minimizing sodium intake and using diuretics to eliminate sodium (unfortunately thiazide, and other diuretics also reduce uric acid elimination and increase magnesium elimination, whose deficiency causes or exacerbates hypertension). On the other hand, it is well known that uric acid increases cell uptake of angiotensin II, which in turn increases cell uptake of sodium urate, causing hypertension.
\n
\n Accordingly, using blood donation to eliminate excess Fe and Mo and taking copper and Mg will reduce urate to ideal levels (3.4<uric acid<4.1), reducing the risk of all the diseases mentioned above.\r\n

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\r\n \r\n Fructose and alchohol consumption increase uric acid while VITAMIN C reduces it. Vitamin C also increases the ability to absorb iron.\r\n

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\r\n \r\n High doses of vitamin C contribute to uric acid load.
\n
\nNettle leaf and juniper berries help to reduce uric acid. Nettle is good for the thyroid as well. Hypothyroidism contributes to uric acid elevation, so nettle plays a double role. Nettle is also good in dissolving both uric and calcium oxalate kidney stones. You just can\'t go wrong with nettle. Juniper berries stimulate the excretion of uric acid. Go easy with juniper berries though. It can lower blood sugar more than you expect.
\n
\nMalic acid is a good way to drop uric acid levels. This is why tart cherries are so good. They are heavy in malic acid.
\n
\nMolybdenum is involved in the conversion of purines to uric acid.
\n
\nAlcohol increases uric acid, because it inhibits uric acid secretion, and beer has high purine content which increases uric acid.\r\n

\r\n __________________
\r\n .
\n- Jim
\n
\n �Men occasionally stumble over the truth, but most of them pick themselves up and hurry off as if nothing ever happened.� Sir Winston Churchill\r\n

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\r\n \r\n
\r\nEffect of Oral Vitamin C Supplementation on Serum Uric Acid
\r\n\r\n

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\r\n \r\n Hi Ted,
\nMost clinical trials are short lived and provide little information on long term results. 400 mg or more vit C per day increase Cu elimination (exacerbating the deficiency we experience with andropause or menopause) and enhance Fe uptake and act as an oxidant (ethanol is also an antoxidant in small doses and an oxidant in higher doses).
\n
\nAs I mentioned, blood donation to eliminate Mo & Fe and supplementation with Cu, Mg & vit D will prevent gout attacks and other conditions. The best additional measures for gout (and metabolic syndrome in general) are avoiding sodas and minimizing ethanol intake, drinking green tea (PQQ, epigalocatechins, etc, and reduces Fe absorption), eating dairy products (lots of protein, Mg, etc, and almost no Fe & purines), celery seed, flaxseed, beets, cherries, white meats (especially fish), taking 1.4g/d fish oil, Si, Cr, PQQ, vit B6, folate, quercetin, etc, and taking a teaspoon of sodium bicarbonate in a glass of water and hot brine baths.
\n
\nDoctors often ridicule bloodletting as stupid, primitive, supersticious and absurd. However, good physicians always knew when to draw blood and when not to do it. They usually did it when it was dark red (high hemoglobin and hematocrit) and never in excess. The idiot who killed Washington hadn\'t a clue and is often quoted as an example of the uselessness and danger of bloodletting, whereas the thousands of people who benefitted from bloodletting are never quoted. Today phlebotomy is the treatment of choice for hemochromatosis, one of the most common hereditary conditions and which may require weekly bloodletting for a year or longer in some cases, until the excess iron is eliminated.
\n
\nIt is interesting that Celsus in his encyclopedia (almost 2 millenia old) states that bloodletting and drinking donkey\'s milk will promptly cure gout, which is perfectly true and coincides with my recomendation to consume dairy and donate blood until 35<ferritin<58. Unfortunately Celsus couldn\'t measure ferritin, but blood banks can, yet don\'t. You can find Celsus\' encyclopedia in latin and english in the web.\r\n

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\r\n \r\n It is interesting that physicians are as affraid of using bloodletting (blood donation) as they are of administering a blood transfusion to an anemic woman with hemoglobin of 9 or even 8 with a copper intrauterine device. Removing the IUD and transfusing 2 units in such women can enhance health enormously in a day, yet doctors sometimes leave the IUD and prefer to administer iron and vit B complex pills that act extremely slowly, while the woman continues losing blood every menstrual cycle. losing much of the blood she made between bleedings.
\n
\nYou probably have heard the expression women are from Venus, men are from Mars. It turns out that in alchemy Mars is the symbol for iron and Venus is the symbol for copper. Young women are often deficient in iron (and molybdenum) and have excess copper, while middle aged men and menauposal women have the opposite combination and correcting the imbalance in either case is most helpful.\r\n

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\r\n \r\n A reduction of 0.35 in uric acid with vit C is by no means significant. Doing what I suggest can easily reduce uric acid from 8 or more to 4.1 of less, that is significant.
\n
\nMoreover, uric acid is measured only in serum. If the uric acid were taken up by the cell (as when angiotensin II is high or during a gout attack), serum levels will drop, but overall stores may be even higher and it wouldn�t show in the study.\r\n

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\r\n \r\n First, and I\'ve stated this before, the only reason women generally live longer than men (by up to 4 years) is because they menstrate, thus eliminating excess iron from their bodies. This is also a good reason to avoid multivitamins that contain iron in any amount.
\n
\nSecond, vitamin C does increase iron absorption, however, this only affects iron absorbed from veggies, which is not a large amount. To put this another way, if you take large amounts of vitamin C daily, such as I, then supplement with curcumin. (400mg twice per day)
\n
\nCurcumin is one of the most important health herbs there is, and should be taken for many reasons, one of which eliminates excess iron in the blood.
\n
\nAnd finally lets discuss gout/uric acid. Of all the research I\'ve seen, there has never been any substantial proof that gout is made by in the liver, nor is it due directly to purines...these are pure speculation with no scientific proof, and is generally found on websites such as Mayo Clinic, which is one site I do not trust, since they have ties with big business/pharma.
\n
\nUric acid is produced by certain species of fungi, and since fungus requires refined carbs for fuel, gout is made worse by consuming sugar, alcohol (fermented sugar) and yeast. (from beer or bread)
\n
\nVitamin C kills fungus.\r\n

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\r\n \r\n It is quite difficult to eliminate iron once it\'s in your body, even pharmaceutical iron chelation agents act quite slowly and while it is eliminated in urine, the Fe damages the kdineys. You claim curcumin eliminates it, I don�t see how. Most people never check their ferritin, have you ever had your ferritin checked? you might be surprised.
\nEach time you donate you eliminate about 250 mg Fe, the equivalent of chelation therapy over months and at a much lower cost.\r\n

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\r\n \r\n PUBMED curcumin iron chelation
\nI agree donation is quicker and cheaper\r\n

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\r\n \r\n Thanks Ted, I had used curcumin for years but wasn\'t aware that it chelated iron, so I�ll be careful not to use it internally in young women and children.
\nHave you ever had your ferritin tested?. After seeing ferritin tests for years I�ve realized that very few people are within the range I suggest. Very many young women and people with internal bleeding have ferritin<30 and most men over 30 and menopausal women have over 100. Mine was 169 the first time I checked it when I was 41 yo (I\'m now 55). I checked it because I figured out that Fe caused gout, which affected all the males on both sides of my family and which I wanted to avoid.
\nOriginally I donated every 2 months and now have no problem keeping it in my range by donating twice a year. My last uric acid value in September was 3.85. I never had a gout attack and cured all my relatives by having them donate & take copper, etc,\r\n

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\r\n \r\n I\'ve found your contributions to the forum very interesting and thought provoking
\nPerhaps you\'d like to ponder this paper and give me your opinion. Aluminium ~ ferritin complex and Alzheimer\'s disease
\n
\nHave your thought about Lactoferrin and raw milk and the role of melatonin in all this?\r\n

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\r\n \r\n By the way correcting low Cu/Fe not only reduces uric acid, but also homocysteine, histamine and superoxide all of which are destroyed by copper enzymes. It also enhances blood vessel and cartilage repair by increasing lysysl oxidase, the Cu enzyme that repairs them and Cu corrects other problems (increases dopamine & melanine which also decline with age).\r\n

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\r\n \r\n Thanks for the article Ted,
\nI am puzzled by the reasoning and the findings.
\n1) People on dialysis are exposed to toxic amounts of Al in order to precipitate out the P, yet supposedly their ferritin has much lower Al than that of AD patients.
\n2) Ferritin also stores Mn & Ni, which can cause major protein misfolding and which were not even considered in the study.
\n3) Ferritin is a metal trap and its contents are not released until the ferritin is damaged or citrate enters it to extract Fe, etc, On the other hand, metal carrier proteins such as transferrin are more likely to cause damage and are not mentioned in the study.
\n4) Would serum ferritin be representative of CSF ferritin, since huge ferritin can hardly cross the BBB? But tiny transferrin can.
\n5) my most absurd comment: tau is known to spread among neurons along axons, perhaps ferritin plays a role in this, acting as a roller.
\n6) Just like Cu&Mo and Cd&Zn displace each other, Al displaces Si, a highly underrated element, which plays an important role in AD.
\n
\nBy the way, an incredible fact, the most common test used to evaluate serum Mg mistakes Al for Mg (Al is included in the Mg reading).
\n
\nYour question about lactoferrin, raw milk and melatonin is quite vague. Like all hormones (including vit D, a secosteroid hormone, which is stupidly called a vitamin) melatonin has myriad functions from sleep and depression to inhibiting myeloperoxidase (production of HOCl and HOBr and hence taurine chloramine & bromamine) and it wanes with age or Zn deficiency.
\nLikewise, lactoferrin is a fascinating substance with crucial roles in infection, cancer, etc,
\nRaw milk I consider too dangerous for consumption, especially since Brucella and TB are rampant in Mexico.\r\n

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\r\n \r\n Another benefit of correcting a low Cu/Fe is that gut uptake of iron is blocked by hepcidin (a Cu protein) and that hephaestin (another Cu protein) ensures adequate Fe mobilization from the liver to the bones, where it is used to make hemoglobin. When Cu is low there is increased Fe uptake in the gut and Fe accumulation in the liver, which is not properly mobilized on demand and there may even be anemia (despite Fe overload). When most physicians detect anemia, they simply prescribe Fe (without checking ferritin), exacerbating the problem.
\n
\nAccordingly, serial, weekly blood donation without Cu, Zn, Mg, etc, supplementation, as is often used for hemochromatosis, etc, or even repeated, voluntary blood donation without supplementating deficient or even moderately low elements can cause major problems. In contrast, complementing phlebotomy with supplementation can enhance health dramatically.
\n
\nFor anybody interested in cell Fe uptake, one of my articles, there is another, older article on Fe, Mo, Cu, etc, in gout, also in medical hypotheses.
\n
\nDo mitochondria regulate cellular iron homeostasis through citric acid and haem production? Implications for cancer and other diseases.
\nJohnson S.
\nMed Hypotheses. 2003 Jan;60(1):106-11.
\n
\nPMID: 12450775 [PubMed - indexed for MEDLINE]
\n
\nI have also started a thread about the Thiazide Holocaust
\n
\nhttps://www.natmedtalk.com/f50/26396-...tml#post209168\r\n

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\r\n \r\n I have found that uric acid levels are not always proportional to ferritin levels. In extreme iron deficiency (iron depletion in Wilson disease, young girls with low Fe intake, etc,) when ferritin can be <4, serum Fe <35 and hemoglobin<10, in its desperate effort to capture Fe, the body captures manganese (Mn) which builds up and replaces Fe in xanthine oxidase, enabling production of uric acid despite Fe depletion, so these people may have urate>4 and even as high as 7. In contrast, people with more common, moderate Fe deficiency (ferritin around 15, 30<serum Fe <60 and hemoglobin around 12) have lower Mn and usually uric acid<3.
\n
\nIn case of iron depletion caused by excess Cu (hepcidin blocks gut Fe absorption), iron supplements are useles and often cause problems. By far the best solution is transfusing 2 or 3 units and administering high dose vit C, penicillamine, Zn and Mo to increase Cu elimination.
\n
\nAlso, as mentioned already, uric acid is not propotional to ferritin levels when inflammation induces production of Fe-free apoferritin, which unfortunately is detected in tests as Fe-replete ferritin. So for example in a lupus patient with a ferritin of 1,000, there may be uric acid<3 and serum Fe<50 and there may be Fe deficiency. If apoferritin could be differentiated from Fe-replete ferritin, onw coul tell the contribution from inflammation.\r\n

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\r\n \r\n I�ve started a new thread about the absurd way lab reference values are determined
\n
\nhttps://www.natmedtalk.com/f24/26427-...tml#post209302\r\n

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\r\n \r\n The Iron in Our Blood That Keeps and Kills Us Interesting article\r\n

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\r\n \r\n Thanks Ted, great article.
\n
\nIsn\'t it incredible that despite hereditary hemochromatosis having been described at the end of the American civil war, doctor Casadaban (a geneticist who should have known that this is one of the most common hereditary diseases) never checked his ferritin and discovered he and his relatives had it?
\n
\nThat\'s why for years I have asked everybody to have their ferritin, cytometry and serum iron analyzed, which together provide invaluable information. Finding even one person with HH can save many in his family.
\n
\nUnfortunately, HH is the tip of the iceberg, even ferritin>80 can contribute to many problems, especially when Cu and Mg are low. Just like scurvy is only the extreme manifestation of vit C deficiency.\r\n

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\r\n \r\n A few years ago, talking to a famous hematologist from Mexico city in his 60s, who had probably seen thousands of patients. I asked him if he had encountered a lot of cases of HH. He said: "Never, that disease si quite uncommon", he simply refused to believe that I had encountered several in a few years, but which saved many more after their relatives also tested their ferritin and started donating.
\n
\nEverybody should check their eye sight, blood pressure, glucose, ferritin, serum Fe, cytometry, urine, etc, at least every 2 years and ideally every 6 months. If everybody did, the cost would be extremely affordable and the data invaluable for the person and for society.\r\n

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\r\n \r\n This small clinical trial for phlebotomy on gout gives you an idea of the benefits. However, the frequent phlebotomy exacerbated the Cu and Mg deficiencies and caused a Zn deficiency. Had these elements and vit D and B complex been supplied, the patients would have faired even better, resolving gout in all of them.
\n
\nhttps://www.ncbi.nlm.nih.gov/pubmed/12832712\r\n

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\r\n \r\n It is interesting that fungus infections (Candida and other fungi, including some causing nail infections) can be so difficult to treat. Many people take MMS, silver, herbs, antimycotics, etc, for years and in some cases succumb to the infection or take them for life, while some people are never affected by fungi.
\n
\nCorrecting imbalances and deficiencies can work much faster and promote health better and more enduringly:
\n
\nEliminating excess Fe and Mo with blood donation or correcting and Fe deficiency when it exsts and taking Cu, Mg, Si, biotin & thiamine (which are depelted by fungi), etc, can boost immunity considerably and make life more difficult for fungi.
\n
\nMany bacteria in our flora produce large numbers of very effective fungicides and fungus growth inhibitors. Unfortunately, our flora can be affected considerably by antibiotics, diet, etc, Which is why fecal tranplantation can help in many cases by restoring production of myriad fungicides, biotin, etc,
\n
\nApelin regulates membrane and blood vessel permeability and is necessary for adiponectin synthesis. When apelin is low, the gut is permeable, increasing the risk of pathogenic organisms and proteins entering the blood stream. When adiponectin is low many inflammatory hormones rise. Apelin synthesis requires omega 3 and glutamine. Unfortunately, glutamine levels decrease either when there is high fat/muscle, when there is aminoaciduria or when ONOO- (a nasty oxidative radical) inhibits glutamine synthase.
\nAccordingly, taking omega 3 and glutamine and inhibiting or neutralizing ONOO- boosts apelin and hence adiponectin, reducing permeability and inflammation and boosting immunity.
\n
\nTaurine is our bodies diuretic and is used to make chloro and bromo taurine, which the body uses to kill many pathogens and regulate immunity. Unfortunately, taurine is often low in kidney disease (amino aciduria causes taurine & glutamine loss in urine and reduced taurine production), liver disease, pancreas disease, etc, So taking taurine supplements can help in many chronic conditions.
\n
\nCarnitine is produced in the kidneys and is required for fatty acids to reach mitochondria so they can be metabolized to produce energy. Unfortunately, its levels may be low. Low cartinitine harms the heart considerably, since it can only process fat (not sugars and proteins as other muscles do), contributes to fat accumulation, bone loss, etc, Since fungi can easily ferment sugars, but have difficulty processing fats, ensuring adequate carnitine levels and increasing healthy fat and protein intake and reducing single carb intake can boost immunity and make life difficult for fungi.
\n
\nChromium+biotin+thiamine and a low carb diet can boost insulin function considerably, resulting in lower glucose levles, avoiding hyperglycemia, which dampens the immune response and favors fungus growth.\r\n

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\r\n \r\n Once again an excellent post that gives much food for thought.
\r\nI came across this today that fits in well with the Fecal transplant story.
\r\nFecal transplants successful in treating intestinal ailment
\r\nIt seems there is nothing new in this idea.\r\n

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\r\n \r\n After watching Dr tents video, Exploding Autoimmune Epidemic, I would be reluctant to have a blood transfussion again, being that blood can carry many virusus.\r\n

\r\n <!-' + '- controls -' + '->\r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n <!-' + '- / controls -' + '->\r\n

\r\n \r\n Ted,
\nI couldn\'t read the article, the link sends me to it but then access is denied. I also tried to search for it in the Los Angeles Times and the same thing happened.
\nThanks anyway.\r\n

\r\n <!-' + '- controls -' + '->\r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n \r\n <!-' + '- / controls -' + '->\r\n